“First my trouble started in my finger tips with throbbing. It
seemed as if the blood circulation was half stopped. My hands
began to swell and I could not wear my rings any more. I had
terrific pains and then I began having trouble with my legs. I
began to have contraction of the muscles, my toes would draw up
into knots. I went to Dr. Baldor about it. He gave me one thing
after another, but I did not improve any. He sent me to the clinic
for the metabolism test, and after that he started a different
treatment. It was some drops. All the time I was taking on like
crazy. I could not sleep at night. My husband had to lift me up in
bed. My hands and legs got steadily worse. Finally I got so bad,
my husband had to pick me up and put me in the car. I could not
get in. My legs would just turn to water. Dr. Baldor gave me the
Koch Treatment. About two or three weeks later, I felt like a new
woman. My strength came back, my legs and hands cleared up, and I
can use them again. I now have a job demonstrating. I carry a suit
case weighing fifty pounds in and out of homes.”
The Basal Metabolism Rate was taken three months after the
Treatment, and found to be perfectly normal, namely plus 6, and
physical examination showed her normal in all other respects, no
sweating, no jerking, no tremor, no muscle twitches. The
exophthalmus had completely disappeared, and so had the thyroid
tumors. The thyroid gland was normal on palpation, inspection
and function. The pulse was normal 80 to 90, and so was the
blood pressure, 140/80. She was strong, slept well, and without
any trace of the former disease.
An analysis and interpretation of this case notes two toxic
states — one that is the result of the forced secretion of the
thyroid cells, the thyrotoxicosis that nearly killed the
patient. The other is the toxin that blocked the regulated
energy production and the regulated energy acceptance and
utilization in both the thyroid cells and the tissues in
general. This is the pathogen toxin, which the Postulate
identifies as an amine of higher O/R potential than the
functional Carbonyl (FCG) group of the tissue cells that could
dehydrogenate and thus burn out of the way. The pathogen
toxin therefore had the upper hand and as it was being increased
in amount, its effects were also increasing as the block to
energy production and energy acceptance by the tissue functional
Carbonyl groups. These normally initiated oxidations that
produce energy efficiently, and received energy in a regulated
way to perform work.
As a result the Krebs Cycle energy production took over, and
had already largely replaced the high efficiency oxidations of
the FCG’s, when the patient came under observation of Dr.
Baldor. If the toxic amine pathogen had been subject to
dehydrogenation at the hands of the FCG, like the usual run of
pathogens and fuel substrates, it would have been burned out of
the way and would not have condensed with the FCG of either the
high efficiency energy producing system, or the FCG of the
energy accepting system of the cell, and blocked their
functions.
It was evident clinically that energy was not reaching the
working mechanisms. This was seen in the steadily increasing
weakness of the skeletal and heart muscles and nervous system.
It was also clinically evident that energy production was going
on at the highest rate shown by the BMR of 104%. It was also
seen that the thyroid gland was forced to the limit in producing
its secretion to push cell activity to evolve more energy. But
no matter how much was produced even to the exhaustion of the
patient, none was used by the energy-starved cells. The patient
lost weight rapidly and to the extreme to supply material for
energy production, but it could not get into the working
mechanism via the blocked Carbonyl groups of the energy
accepting system of the Postulate. The basic pathology then was
the block to energy acceptance by vital working units. Since
one toxin, an amine of high activation was the pathogen, it is
also evident that the FCG’s of energy production and energy
acceptance are similar atomic groups and since these are
dehydrogenators, the Postulate identifies them as highly
activated Carbonyl groups. This conclusion is supported by the
type of response to oxidation the integrated toxin gives, after
it is condensed with the tissue cell FCG’s. That is, the type of
cleavage observed is that of an azomethine double bond when its
alpha positioned hydrogen atom actually invites dehydrogenation
and is removed so a free radical can be formed and add molecular
oxygen to become a peroxide free radical which, accomplishes the
oxidative separation with restoration of the functional Carbonyl
groups of the tissue cell and the toxic amine group is burned
away. The facts of the case history support this explanation.
(See Appendix).
It is seen here that the pathologic state actually invites
correction, and any clinician would suggest correctly how it
could be done. He would say, since the FCG cannot
dehydrogenate the toxin and start its combustion, because its
O/R potential is too low, then the thing to do is to offer a
Carbonyl group of higher activation with a potential equal to
the job. This is what was done in this case. A molecule
of correct steric advantage carrying a Carbonyl group of high
O/R potential was used. The results were the rapid reversal of
the pathogenesis. As soon as the integrated toxin was burned out
of the way, energy could enter the cell working units, and the
urgent call for more energy stopped. The thyroid was not called
upon to whip up the tissues to do more oxidizing, and the
nodules it had developed to aid its work subsided and
disappeared. The BMR dropped to a normal of plus 6%, and all of
the symptoms of the thyrotoxicosis, and of the basic pathogen
disappeared. The woman was normal in 3 months after one dose of
a highly activated Carbonyl compound.
This case proved a few things in the Koch Postulate, and
it also shows that the thyroid secretion takes no part in the
oxidation process, any more than the poisonous nitrophenol
series, that some have classified as accelerators of the
oxidations. As we pointed out here, the thyroid function is to
whip up the cells to put their oxidation apparatus to work to
supply the energy needed for the occasion. It itself does not
enter the oxidation process. Nitrophenol blocks various
esterifications with phosphoric acid which normally form high
energy carrying phosphate bonds. Thus it starves the cells of
energy and the tissues are whipped up to produce more energy for
survival, just as in the case at hand. Nitrophenol thus works as
an “uncoupler” and is so classified. It prevents the energy
accepting mechanisms from receiving the energy. In the case at
hand, the energy came to the doors of the energy accepting
mechanism, the FCG of energy acceptance, but the door was
closed, — blocked by the condensation with the amine compound.
Thus the Carbonyl group of energy acceptance was already
occupied and could not condense with the amine of the ATP that
carried the energy that would be liberated by ATP-ase with the
help of calcium. Our Postulate goes on to explain that with
the liberation of energy by the hydrolysis of the ATP to ADP,
the phosphoric acid set free can split the azomethine bond
setting the ADP free to again do another cycle of energy
transport with the acid.
One sees that there is no similarity in the actions of the
thyroid secretion, the nitrophenols and the highly activated
dehydrogenator Carbonyl compound (SSR) used to oxidize the
pathogen out of the way. The SSR actually took the leading part
in the oxidation mechanism and did the work the normal oxidation
initiator would have done, if it had an adequate O/R potential.
It is not possible to compare a reagent that prevents energy
storage for use in work, with an agent that produces energy for
use in work, and besides, actually starts the oxidation process
in the cell by burning away the pathogen that was blocking
energy production.
Further, the nitrophenols are pathogens whose action can wear
out, if not forced too long. But if they are forced too long,
they are subject to reduction to aminophenols, which would then
act much like the pathogen in this case, and block the
initiation of the oxidation progression, and bring about a
dangerous situation much like in the case at hand. The
nitrophenols proved to be pathogens in the attempt to beautify
obese patients. The reduction in weight took place but, in too
many, the destructive action continued because of the situation
that existed in the case we are discussing, and these victims
went on to their deaths. They need their FCG’s freed from the
obstructive amine as was accomplished in this case. However, the
experts are still at sea with regard to the true action that
caused the fatalities.
The block in the use of the energy of oxidation by
dinitrophenol is seen also in its inhibition of mitosis in Sea
Urchin eggs reported by Clowos (1951 Ann. N. Y. Acad. of
Science). Even though the dinitrophenol in doses of .01 mm
concentration caused a fourfold increase in the consumption of
oxygen, the mitosis and phosphorylation was cut in half, and
further increase in the concentration of the poison completely
blocked mitosis and phosphorylation. So whether the oxidation
process is blocked in producing energy, in transferring and
carrying energy in phosphate bonds, or in receiving this energy,
the reactive response is to produce more energy to make up for
the energy starvation, in the tissues whose working mechanisms
do not receive the energy. Thus an analysis of effects of toxic
amines and nitrophenols shows they do not give impetus to the
oxidation mechanism, but block its ultimate purpose—the supply
of energy to the vital mechanisms of the tissues. Here we find
in 1951, a nice confirmation of our Postulate measured with
microscopic accuracy.
The thyroid secretion is a hormone whose intimate action is
still unknown. However, it does not take any part in the
oxidation process itself. Comparing its action with that of the
SSR, one sees that the latter took the leading part in the
oxidation mechanism. Further the action of nitrophenol and of
thyroid extract are of different orders and challenge
comparison. The former always has a toxic action, the latter is
physiological, but the action of both, as explained before, is
very different from that of the SSR. The high BMR in the case at
hand, has a pathologic cause depending on the pathogen that
blocked energy acceptance by the cell’s working mechanism.
The statement of some biochemists that the oxidation process
has no immunological significance is based on the fact that
the Krebs Cycle has none. We gave the reasons before. The
O/R potentials of the participants are too low. Then these
biochemists also hold that the Krebs Cycle is the only mechanism
concerned in the tissue oxidations, and is all sufficient. They
do not consider that the Krebs Cycle is a hang-over from the
process used by primitive forms as bacteria and though it is
retained by the higher forms as animals and man, it is only
used by such as an alternative pathway, when the High
Efficiency System already explained is inactivated for a time.
That it offers no protection is seen. Moreover, it gives
no clues to the explanation of the Pasteur Effect. The early
Chapters of “The Survival Factor in Neoplastic and Viral
Diseases” show how both depend on the action of the FCG.
While the Carbonyl group that initiates the oxidations of the
High Efficiency Smokeless System, lacks the high O/R potential
Carbonyl dehydrogenator, that some pathogens require for their
destruction, yet its range of O/R potentials is twice as high
(0.7 v) as that of the Krebs Cycle participants (0.3 v). So the
opportunity to give protection by the High Efficiency System is
considerable, — enough to maintain good health under the usual
circumstances. The use of a Super-high Carbonyl dehydrogenator
of correct steric advantage is proven, in this case, to offer
protection by way of an oxidation process that imitates that
Postulated for the High Efficiency System, and a close analysis
of this case is all that is needed to prove the existence of the
High Efficiency System.
However, two more cases will be submitted to show that the
toxic basis for malfunction can be removed, and the pathology
corrected by the processes of adequate dehydrogenating
efficiency, started by the Super-high dehydrogenator, and
continued by the natural dehydrogenator (FCG) system.
POST-PNEUMONIA NEPHRITIS
CASE No. 2
Dr. Evans
Tom E. 4 years old, was recovering from bilateral
bronchopneumonia, when suddenly he took a convulsion of
considerable severity. Oedema rapidly developed with blurred
vision, headache, dizziness, delirium, etc. The urine secretion
diminished as the oedema rapidly increased. The blood pressure
was found to be 146/68, and the blood non-protein nitrogen 74.6
mgms %. Twelve hours later the pressure rose to 160/100, and two
days later it was 180/130 showing a rapid development of the
pressor substance that blocked the kidney elimination. The
oedema had developed by then to the point where the contours of
the chin and neck were obliterated, and very little urine was
passed. Then the second convulsion took place. It was severe and
the boy passed into coma. It was in this condition that he
received the Synthetic Survival Reagent. A few hours later, the
mental symptoms had improved, he came out of the coma; soon the
headache, blurred vision, delirium, etc., gave way to rational
mental comfort, the blood pressure steadily dropped and the
urine increased as the oedema disappeared. The blood pressure
was found normal in a few days with a normal non-protein blood
nitrogen of 25 mgms.%. The correction was completed by rescuing
the FCG so it would go back to work again. The pressor substance
is well known now to be a toxic amine, so our Thesis is
supported nicely by this case also.
ECLAMPSIA
CASE No. 3
Dr. Baldor
Mrs. D. was married seven years and could never carry a baby to
term. Abortion was required before the end of the second month
of pregnancy each time, and the period was shorter each time.
This was the 4th pregnancy, and they all followed the same
course and symptoms but with increasing severity. In each
instance she vomited profusely, with much salivation constantly;
the urine was progressively decreased until only blood came,
just as in the last hours of the parathyroidectomy intoxication.
Convulsions followed by coma called for immediate abortion, if
life was to be saved. This time, however, Dr. Baldor tried the
Synthetic Survival Reagent as her big ambition in life was to
have a baby. Twenty hours after the injection was given,
vomiting had decreased from 20 times a day to twice per day. The
urine increased and, in 72 hours, she was passing half a liter a
day. This urine still carried blood and albumen. In four days,
she passed a full quart of urine per day. The vomiting
disappeared entirely within two weeks, but the salivation had
continued and, during the third week, vomiting started again.
She was given another dose of the Survival Reagent, and all
cleared up quickly thereafter. No more symptoms of eclampsia
returned. She carried her baby comfortably into the seventh
month, when she had an automobile accident, and spontaneous
abortion threatened, so she was delivered of a 5½ pound baby
that thrived well. She had no return of eclampsia symptoms and
gained full health quickly.
Here we see again that the toxin that blocked the oxidations of
function and the regulated energy acceptance by the working
mechanisms, could be removed by an atomic group similar in kind
but of higher O/R potential. The allergic uncontrolled spasms of
the small blood vessels, and the anoxia caused by colloidal
gellation, had to yield to restored efficient FCG function. The
basic pathology was met and corrected, at its very inception. Still,
some of America’s greatest biochemists and clinical experts
claim that “the oxidation mechanism has no significant action
or position in the maintenance of health or in the combat
against disease.” They are limited, of course, by the
performances of the Krebs Cycle, which to them is the whole
oxidation mechanism. But, if one were to accept such a dictum,
one would have to add, “it is impossible to die of asphyxia.”
The predicament is rather contrary to progress.
TOXIC GOITRE AND CANCER OF THE STOMACH
CASE No. 4
To show that one toxic agent (removable by one corrective
attack) can cause a toxic hyper-function as in Case I,
and also cause a very high-grade malignant neoplasm of the
stomach in the same person and at the same time, the case of
Mrs. W. is offered.
At the time this patient was treated, the Geiger Counter had
not yet been invented, so it was impossible to estimate the
earth’s irradiations in her environment. However, it is noted
that she lived in what is known as the goitre belt, a region of
iodine deficiency and also of high cancer mortality rate. Her
daughter had been treated for a rapidly developing brain tumor.
Many other patients came from this region for Treatment.
However, one thing this study lacks is a systematic correlation
of the terrestrial radiations with cancer incidence and also the
number of conditions allied to cancer to be met; and most of
all, how the terrestrial rays affect the recovery rates both of
the neoplasms and of the allied diseases.
There was no history of cancer in the ancestry, but her husband
died of cancer 8 years previously, and her daughter, with a very
malignant tumor, was only 28 years of age as compared with the
patient’s age of 58 years, at her first visit. One recognizes
here the vigor of the carcinogenic flux of this region. Both the
mother and daughter made typical recoveries under the Treatment.
There was nothing in the geophysical environment that interfered
with the cyclic reactions and the steady progress of the
recovery process. One feature to be noted is that as cancer is
associated with aging processes, this patient, at only 58 years
of age, looked like a person twenty to thirty years older. The
skin and tissues in general were senile, though the hair was not
grey. During the recovery process the senility changes
disappeared. The main features were as follows:
The disease started two and one-half years previously as a
steadily increasing nervousness, progressive cardiac weakness,
tachycardia, increasing ease of perspiration, loose bowels, and
tremor of characteristic hyperthyroid type. Radiographs showed
considerable enlargement of the heart and mediastinal shadows
early in 1927. There was dyspnoea on slight exertion or lying
down. Exophthalmus developed rapidly, the skin was bronzed, and
gastric distress and inefficiency set in. The feet and ankles
swelled considerably, yet she lost weight, falling from 150
pounds to 108 pounds in less than nine months.
The physical examination revealed the exophthalmus as shown in
the photograph before Treatment. There was also a greatly
enlarged lymph gland (walnut size) in the left supraclavicular
space; the veins of the head and neck engorged with blood when
she laid down, and percussion showed a marked increase in the
mediastinal dullness.
Examination showed the epigastrium and the whole area below the
costal border down to two centimeters below the umbilicus on the
right side to be occupied by a huge, bulging, solid, fixed,
irregular tumor. The stools showed decomposed and occult blood.
There was vomiting and great weakness and considerable pain
throughout the abdomen. Thus the stomach, the liver, and
probably the suprarenal glands were involved by the neoplasm. At
the time of this examination she was very weak.
Mrs. W. before Treatment showing the exophthalmus from toxic
goitre excited by the carcinogenic toxin. (Left photo)
Mrs. W. after Treatment and recovery from cancer of the
stomach, and toxic goitre as secured from one chemical reagent.
The exophthalmus is gone for good. (Right photo)
One dose of two cc. of a 10-(12) solution of the (SSR) serial
Carbonyl system was used on September 28, 1929. The recovery
process exhibited the usual cyclic three-week reactions, with
chills, fever, and general aching, and with improvement following
each reaction until the recovery became complete. At last report,
ten years later, she was in good health. We lost track of her
thereafter.
Regular FCG function, both for producing and using ATP energy,
was blocked and this showed for the thyroid cells, the stomach
growth mitotic mechanisms and the general tissue oxidations as
demonstrated by the senility changes. Still the Krebs Cycle
oxidations went on, and fermentation supported the neoplastic
cells. Had we supplied a Carbonyl group of FCG oxidation
potential, we probably would have gained nothing. However, a
Carbonyl group of boosted O/R potential cleared the inactivator
of FCG functions away so normal FCG metabolism (in contrast with
the Krebs metabolism) was restored, senility, toxic goitre, and
cancer all faded away permanently.
TOXIC GOITRE AND ETIOLOGICAL TOXIC FOCI
CASE No. 5
Dr. Jayme Treiger
In this case the pretreatment control or observation period
lasted from September 12, 1953 until March 13, 1958. The
development of the etiological factors with the progress of the
disease itself was well noted.
Mrs. D. S., F. 27 years old, married, a thin brunette woman,
very nervous, complained of dyspnoea, cold sweating, pharyngeal
spasm (sensation of an egg in her throat), able to bear heavy
duties but not simple ones, urine sometimes fetid and strongly
colored, acne, leukorrhea, sometimes bloody, and painful nodules
in the right breast. These breast symptoms arrived after a
second electro-coagulation of an ulcer on the cervix uteri,
produced after the second childbirth. These nodules were helped
by hormone treatment for a while but had returned, with further
toxic symptoms as a tachycardia of 106 per minute, and slight
thyroid enlargement, that started nine years previously. She had
pertussis, measles and vericella during childhood.
During and since childhood, she had periodic crises of angina
with high temperature and pus from the tonsils. Homeopathic
treatment helped the tachycardia and the throat spasms and made
her feel much better, but the basic pathology was not retarded,
and she went to a gland specialist who treated her from November
1954 to January 1956. From him she received Dexamyl, Somniphene,
Prometron, Ovocycline, Diiodotyrosine, Apliotil, Thiouracil, and
Nodular on different occasions. She did not improve on this
series of modified benzene rings, though enough were tried. This
shows that the therapeutic conception was not based on
physiological considerations, but was the fruit of modern
pharmacology.
Feeling worse, she returned to Petropolis. The B.M.R. by Dr. T.
showed a plus 45 and Cholesterol of 122 mgms. % on 3/12/58. She
was now exhausted, extraordinarily excited, always tired,
difficult to sleep, with frequent nightmares, pulse 106 per
minute, and her blood pressure in a low range. She was given 2
millimicrograms of the SSR intramuscularly on March 13, 1958,
and the reactions that followed are indicative of the sources of
her toxins.
Reactions: Tonsillitis that was suppressed from activity while
under the phenolic treatments mentioned above, started to be
active with high fever, pus discharge and pain in violent
periodic crises. The bloody drainage from the cervix uteri that
was suppressed by the cautery started up again. However, one
week after the Treatment in spite of the strong angina crises,
she was feeling very well, as if with renewed vitality. A few
weeks later she reported again. The pulse was normal, 82 per
minute, the blood pressure normal 120/90, and as her good health
was being restored, old symptoms of years of little difficulties
returned briefly and disappeared. She felt good enough to not
need a doctor. The throat had normalized and the cervix uteri
had healed, and she did not return for more observation. She had
received two injections of the SSR, the second one a year after
the first, for while the cervix showed no abnormality on
examination, there were symptoms suggesting reaction in the deep
scars within. The BMR in February, 1960 showed 6% over normal,
the breasts, tonsils, uterus, nerve responses were normal,
temperature 36.7°C, pulse 60, the B.P. 110/70, and she enjoying
the best health she had ever experienced.
In this case the etiological lesions that brewed the toxins
that attacked the breast tissues and the thyroid gland were
respectively the cervix infection and the tonsil infection. The
cautery sealed up the drainage facilities, and made the scar
tissue that was infected more anoxic. The reactivity of the
reticuloendothelial cells of the tonsils to their contained
infections was suppressed by the phenolic derivatives, so the
thyroid was poisoned all the more. Further, the poisons from the
cervix and those from the tonsils while showing some specificity
to the thyroid and breast tissues were also general poisons and
affected all of the tissues making her nervous and weak aside
from a special thyroxin effect. Here the relation of the
reactions (following the Treatment with the SSR Reagent, which
were severe) to her improvement in tissue function showed that
these reactions were not of a vaccination nature, but were
actual reticuloendothelial battles against the disease agents
going on in conjunction with the chain oxidation of these
agents. Then, too, as the various FCG units were liberated from
combined toxins and went back to work, she started to feel
normal and her various functions behaved normally again. It is
to be recalled that after the SSR was given the tonsils became
acutely inflamed, and the cervix lesion broke loose with a
strong inflammatory process. Thereby both lesions were cleared
of their imprisoned germs and fibrosis integrated pathogens, and
as the induced oxidations burned the pathogens away, the
fibrosis disappeared also. The anoxic centers were wiped out, so
the disease was cured right at its very inception. She has no
more sore throats nor cervix troubles, and no more secondary
effects, as thyroid enlargement or abnormal function. The
breasts have no more nodules either. Her general health is
normal. Her nervous system is steady. She sleeps normally and
does not sweat as she formerly did. In other words, the
pathology was fully reversed and discarded. This same course
will be seen in the other cases reported here, and in all others
when one takes the trouble to thoroughly check the recovery
course.
