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SURVIVAL FACTOR IN NEOPLASTIC AND
VIRAL DISEASES
By
WILLIAM FREDERICK KOCH, Ph.D., M.D.
Chapter 8
ATROPHY, ANAPLASIA, AND NEOPLASIA
One may compare the correction of nerve atrophy and neoplasia
very nicely in retinal cases where cell reconstruction can go
on, but not cell reproduction; we may also compare the
corrective responses of anaplastic and neoplastic tissues in
reproductive cells. In all four situations, the correction
is had by restoring normal FCG function and thus a Least
Common Denominator is established in these supposedly widely
varying conditions. So the basic pathology is the same. There
is a suggestion that the requirement for neoplasia is the loss
of the functional mechanism.
In Glioma, there is no development of functional mechanism,
because the cells are too embryonic and immature or
undifferentiated. In the Sympathicogonioma case, one sees how
the nucleus is separated from the functional mechanism, the
fibrillar structure. The latter lays separated as a fibrillar
syncitium supported by cytoplasm, while the reproductive portion
is separated off as individual entities, — cells. Thus the
functional mechanism is discarded. The return to normal which,
we are not able to see must be like the maintenance of the
normal; a correct balance and interaction of the reproductive
and functional mechanisms. Thus the energy production in nerve
cells must serve their work of repair and Nissle substance
synthesis only to keep them normal. But when these systems are
blocked, the energy has only one place to go and that is
mitosis. When the working part is shed as in Sympathicogonioma,
of course, there is the visible separation. The very high-grade
malignancy of such neoplasms may therefore be understood.
In this case of Neuroblastoma, which will be detailed a few
pages further on, visible separation of reproductive and
functional elements of the cell is demonstrated, even though the
child is born with the neoplasm, latent in his system. In the
Retinoblastoma case, the functional mechanism did not exist in
the cells that went malignant, so the mitotic mechanism had no
other course than to use the energy produced in the nucleus for
reproduction. There was no other path for it to follow. It
appears from these two cases alone, that energy production is
primarily evolved in the nucleus, even though the nucleoprotein
grana are found throughout the cytoplasm. In the Glioma case,
these granules did not exist, while in the Neuroblastoma case
their connections with the nucleus were severed.
In the Optic atrophy case to follow, complete atrophy was not
possible as yet, as a result of the Retino-choroiditis, for
regeneration of the affected cells took place with restoration
of function. A bacterial origin, of the toxic injury, is of
concerned here.
There is another situation due to toxic injury, that of chronic
alcoholism, where this weakness can be transmitted from parent
to child for several generations. A case of Delirium tremens
presented on this topic, should interest the geneticist, in view
of the foregoing remarks, for indeed hereditary transmission is
concerned in all of these conditions. Even in this case
of Delirium tremens, the effect of a toxin on the cerebral
cells, the connection between the genes and the functioning
elements is concerned, — the nucleus and the mitochondria.
Evidently, this is the toxin that caused the cancer in this
case, for the Delirium tremens returned during the 12th week
reaction in his recovery from cancer of the face. The patient
himself had never used alcoholic drinks but his father, and
other predecessors, did drink themselves to death. Still, the
patient gave the D.T. symptoms during the correction of the
neoplastic defect. Both defects were corrected by the Reagent,
which corrected the hereditary Neuroblastoma defect, in the case
to follow very shortly.
This case, with its high malignancy characteristics, teaches
another very important fact; namely that trauma, with its
circulatory injury and consequent anoxia alone, is not
sufficient to cause cancer. Also it teaches that the recovery
from this disease removed the carcinogenic factor that requires
anoxia to become effective as our Postulate outlines. For as
will be seen this boy, after being cured by restoration of his
FCG Survival Factor, did not again get cancer after being
severely injured by the automobile accident that bruised him at
the site of the former rumor. This case repeats the lesson
learned from case No. 11 to follow.
Recovery Reactions
Cyclic reactions: 1) often characterized by the symptoms
of the pathogenesis showing up in the reverse sequence to
their coming, 2) accompanied by a local congestion at the
lesion, 3) as well as constitutional symptoms of grippiness,
as occurs in so many virus infections, are to be expected
periodically, while recovery is in progress. The
cycles run in a definite periodicity, in which a three-hour
unit, or more often, a twelve, twenty-four, thirty-six,
seventy-two, or eighty-four hour interval, is usually observed.
Thus a reaction of chills, fever and achiness can show up
twenty-four, seventy-two or eighty-four hours after the
Treatment. Or, it may come the third week, the sixth week, the
ninth week, or the twelfth, twenty-fourth, thirty-sixth,
sixtieth, seventy-second, eighty-fourth, ninety-sixth, hundred
and eighth, or even later multiples of twelve weeks.
The local features in the lesion are: congestion, swelling,
hyperasthenias, hyperreflexia, more or less pain, local heat,
and maybe some bleeding. But this passes off in three hours or a
multiple thereof, and improvement is then noted. When
biopsies are taken of the lesion undergoing recovery in this
way, it will be found to undergo: 1) a coagulation necrosis,
2) then a calcification, much like accompanies or mediates
the digestion of a blood clot or of milk, 3) vascular
in-growth is observed, first angioblastic and fibroblastic
tissue, and 4) mast cells or other white blood cells to help
carry off the debris. (Koch, New York, Medical
Record, October 30, 1920), 4) Angioblastic
tissue finally replaces the growth, and then 5)
functionating parenchyma grows in to reform the organ on
physiological lines. The cure is therefore
complete, as it could not start without preliminary
elimination of the pathogen, and its functional status is
returned. The sequence of events during reconstruction is
exceedingly interesting and will be discussed.
PRIMARY ATROPHY OF THE OPTIC NERVE AND RETINA SEQUEL TO
SCARLET FEVER
CASE No. 6
R. J., 14 years old, gave a family history negative to
tuberculosis and cancer. The pretreatment control period and
diagnosis are well described in the correspondence between the
Henry Ford Hospital and Jennings Hospital experts as follows:
HENRY FORD HOSPITAL
DETROIT, MICHIGAN
Henry A. Du----, M.D. September 10,1946
7815 E. Jefferson Ave., Detroit, Michigan
Case No. 453242 Re: R. J.
Dear Dr. Du----:
Our first contact with the above named child, according to our
records, was a precamp examination done by Dr. J. A. Jo---, of
the Division of Pediatrics, in July, 1945. At this time his
vision was recorded as being 20/20 bilaterally. He was seen by
us April 15, 1946, at which time he had developed a
scotoma in the right eye. Vision without correction on the right
was 8/200, left 20/20. External examination of both eyes
revealed them to be normal. Funduscopic examination of the right
eye revealed a normal lens. In the macular area there was a
chorioretinitis which fits the description given by you in your
letter. The left eye was normal to Fundascopic. Tangent screen
examination was done which showed an absolute scotoma in the
supracentral region. The periphery was normal; red field was
reduced.
He returned to Pediatrics for a general physical examination by
Dr. Jo----April 17. Dr. Jo---- noted that the child had been
seen by Dr. Ca--- who had applied .1 mgm. of 1/10,000 O.T. He
had been negative to 0.01 mgm. when seen by Dr. Jo---- the
previous year.
Sinus and chest X-Rays were made. Dr. Do--- reported them as
showing chronic pathology of both antra and probably the
ethmoids and frontals as well, and were suggestive of a pan
sinusitis. There was only a moderate increase in the
broncho-vascular markings in the bases.
Blood count showed on 4-16-46 a hemoglobin of 13.5, white
blood cells 10,100, red blood cells 4.66, polymorphonuclears 46,
small lymphocytes 52, monocytes 2.
He was last seen April 20 by Dr. Di---- for ear, nose, and
throat consultation. No foci of infection were found in the
ears, nose or throat to account for his eye condition. Dr.
Di---- reviewed the sinus X-Rays and washed out the left antrum.
The return flow was clear.
He did not return for follow-up eye appointment, and we have
not seen him since. Trusting this information will aid you in
your studies, we are,
Sincerely yours,
Henry Ford Hospital
Per; /s/ E. L. W.----, M.D.
M. W. S---, M.D. Surgeon-in-Charge
rms. Division of Ophthalmology.
Our History
Our examination made April 26, 1946, gave a history of
headaches and chronic sinus drainage with acute exacerbations,
sore throats often, and a history of exposure to scarlet fever
that did not take on him. He was slightly sick, but had no rash
and did not peel. He noticed the eye condition when at a
shooting gallery he aimed a gun using the right eye and found he
could not see with it. Thus in one year exactly, a 20/20 vision
went to zero so far as practical use was concerned. He could not
make out objects with it nor read any size print. The area of
“absolute scotoma” noted by the experts was an area of retinal
and optic nerve “atrophy” that involved the upper half of the
retina and the central field. There was some peripheral vision
but it did not help much.
We gave him a 2 cc. injection of the 10-(12) solution of the
serial system of Carbonyl groups (SSR), the Synthetic Survival
Reagent, on April 26th. The sinus drainage and headaches soon
ceased. The sore throats returned only twice during two reaction
periods. But the other symptoms were aggravated during the 12th,
24th, 36th, and 60th weeks. During this last reaction the throat
was most sore, and he had a fever of 103° F. as well as a
typical scarlet fever rash that lasted only 12 hours. His health
was perfect after that.
Examinations of the eye by the same experts found that the
vision in the right eye was 20/400 in August 1946, that is four
months after Treatment. Thus a definite improvement had taken
place. In June, 1948, it was 20/100, and in October 27th, 1948,
it was 20/40. Thus the improvement was continuing. Further
reports, but without examination, claim his eye is perfect so
far as reading fine print is concerned or for any other use.
Here we see the polymerized scarlet fever toxin causing a
degenerative change, and after it was broken down to the
monomeric phase, it gave rise to the symptoms of the acute
infection transiently. This was burned away in only twelve
hours, and the recovery was complete. Other similar occurrences
could be reported.
ACUTE CHORIORETINITIS SEQUEL TO PELVIC AND TONSIL INFECTION
CASE No. 6A
Dr. Jayme Treiger
Mrs. L. P. S., age 35 years. April 9, 1957 when first examined.
Her Pretreatment Control Periodof observation extended until
December 5th, 1958, during which time she received all of the
homeopathic remedies, and a course of Cortisone and its
derivatives with the most recent and efficient antibiotics.
There was a steady downward course in her general health and her
two chief areas of infection, the pelvis and the tonsils, and
steady deterioration of the function and pathology of the retina
of the left eye, that developed during the last six months of
this period.
Physical Examination on entrance showed, fever of 38.6° C. with
a bilateral tonsillitis that had persisted for the past three
years, worse at the end of the menses. She was very exhausted
because of excessive use of antibiotics and antipyretics. Also
there was an inflammatory process in upper and lower abdomen,
sequel to violent gynecological infection sustained shortly
after marriage. The tonsils—red, hypertrophied; tonsils full of
pus, Heart—O.K., Gall Bladder sensitive XXXX, Chaufford Zone
XXX, and Ovaries inflammation. The menses were prolonged. There
was no benefit from medication followed during the first six
months. Indeed headaches, fetid leucorrhea and varicose veins
were aggravated. Three months later oedemas of feet and hands on
waking, and of the upper eye lid with severe sacro-ovarian pains
were added to the symptoms, with severe chest pains, and blurred
vision, also cracking of the skin on the palms of her hands.
Fundascopic examination by the ophthalmologist showed no retinal
pathology at this time, 7/18/58. The examination on 10/28/58
showed the left eyelid congested, vision blurred, photophobia.
Fundascopic examination on 11/5/58 showed left eye vision 0.67
fundas turbid with exudate, and a focus of Chorioretinitis in
evolution, juxta-papillary. Right eye normal. V--I, Signed Dr.
A. H.
Prognosis — Healing with scar causing corresponding scotoma.
This Ophthalmologist placed her on Cortisone and its
derivatives, antibiotics and topical applications. One month
later the eye was strongly congested, much worse than a month
previously, and she could see nothing at all with the left eye,
much worse than at the beginning when she still had some vision,
Her weight was 80 Kilograms. Two millimicrograms of the SSR were
given intramuscularly, 12/5/5 8, by Dr. J. Treiger.
Post Treatment Period-12/6/58, swelling of peritonsilar lymph
nodes, dizziness, uneasiness, and violent pain in the pelvis
recalling the time of her acute initial pelvic infection, with
exquisite tenderness. Vision more blurred for 2 days. Copious
flow of pus from genitals for the past three weeks, 12/31/58. On
January 12, 1959, she had severe tonsillitis that lasted for 3
days at the end of her menses, fever 39.5° C., great hypertrophy
of the tonsils, weight down to 74 kilos. with terrible vaginal
itching, that soon improved. By the middle of 6th week, 1/14/59,
much better, temp. 36.7° C. On 2/17/59 her weight was 72 kilos,
feels better, and shows cracking in the skin of the palms of
hands as of ten years earlier, and skin desquamation. Temporary
improvement of left eye vision. On 3/18/59 she reported she
spent a wonderful week, including her eye. On 5/5/59 she
reported she had two weeks of normal vision and was in good
health generally. On 4/9/60 she had three days of reaction with
slight swelling of the left tonsil, pain in the left eye, slight
blur in vision, and then quickly normalized and has stayed well
ever since. The pelvic and throat pathology completely
disappeared. This was the 69th week reaction,
Fundascopic examination by Dr. B., reads as follows:
Dr. A. De S. B.
Petropolis
Estado de Rio de Janeiro
December 21, 1959
Dear Dr. Treiger:
The “Fundus Oculi” examination which was done on your
recommended patient, Mrs. L. P.S. accused on the left eye an
inferior nasal focus, entirely healed of chorioretinitis all the
way well into the periphery. There is no abnormality concerning
the vascular supply. The visual field through comparison
showed no abnormality. Therefore I did not try the instrumental
perimetry.
Yours truly,
Dr. B.
Discussion -- No scar was produced as always happens
with corresponding blindness. The retina was restored. If or not
a scarlet fever infection played a part in the etiology no one
can say. However, the cracking of the skin of the palms of the
hands and the desquamation that showed up as she finished her
recovery may have some indicative value. Even though it may not
be scarlet fever, it is a toxic effect of infection antecedent
to the retinal lesion. In contrast to the former case which
showed no inflammatory change reportable, but only the atrophic
changes here the inflammatory changes predominated and the
degenerative effects were also in evidence. This woman was also
too heavy at first. She lost weight down to 70 kilos and then
got thinner without losing more weight, showing a building of
solid tissue with functional efficiency that went with her
return of happy good health. Both foci of pus infection cleared
up, and the non-pusy inflammatory and degenerative changes
likewise disappeared. Thus an integration of the toxins of
bacterial infection that excite no leukocytosis, as they are
bound to and become part of a different tissue, are separated
from their host cells by the SSR as we will show viruses are
separated and carcinogens are separated, leaving the host cell
in good functional status while the pathogen is destroyed. This
accompanies destruction of unbound toxins that excite
leukocytosis in other parts of the body.
Her reactions occurred typically, twenty-four hours
after the Treatment was given and at multiples of three week
intervals thereafter for a few times only, the sixth and the
69th weeks. The last reaction occurred after the retina function
had returned to normal and examination showed it was
structurally normal. Thus the reactions are not to the
lesion, but to the cause of the particular lesion, a matter
much deeper than the toxic factor of the etiology. One
case alone demonstrating this phenomenon, would be good to
remember, but many such cases showing reactions for example
coming five years after a large cancer of the stomach has been
absorbed and health follows a normal course, indicate that a
phenomenon is observed that clears up the predilection to the
pathological response to the etiological factor rather than an
attack on the pathogenic factor which is long out of the system.
The correction is more basic than getting rid of the etiological
agent and healing the lesions.
Confirming this, one recalls the case of cancer of the face of
Dr. M, of a small town in Texas in 1927. He was 81 years old and
blind with bilateral retino-choroiditis of several years
standing. He was treated with the SSR for the cancer and while
that lesion was healing up, the vision returned, so that before
the growth had entirely healed, his vision had so perfectly
improved that he could see the string that was used to tie up
packages in the country store that hung from the ceiling. This
was visible to him as he entered the store. He had reactions
which he reported long after he considered himself cured. There
was high fever, 106.5° F. three years after full recovery.
During this fever, which was different from any he had seen in
his long practice of medicine, he was surprised by a good
appetite and the desire to work and expend effort. Here is food
for thought. He reported the best health for years afterward.
Here are three different etiological factors, scarlet fever, a
mixed infection of the gonococcus with strep and staph resistant
to all latest antibiotics, and the etiological factors in
cancer. In the latter case, an infiltrating type of basal cell,
solar rays were one big factor and high winds, frost bite, etc.
that caused a circulatory injury, anoxia, etc. This circulatory
injury was present in the mixed infection case too as scar
formation was in progress with the vascular injury that goes
with the inflammation, and neuron injury. In Case 6, the
inflammatory changes were a minimum but the degenerative changes
were neuron loss and scars were most prominent. This is rather
strange as there was no inflammatory history, but a very rapid
development of the degenerative change. The other cases of this
disease, we may review, add nothing to the present meager
information. This is unfortunate, since the presence of
degenerative change away from the pus forming areas that
supported the infection and toxin production gives one a chance
to investigate the cause of degenerative disease. If we had
sufficient data, we could demonstrate its different steps. This
much is brought to mind anyway, it is the job of an
Ophthalmologist.
The old doctor had plenty of contact with scarlet fever, and he
no doubt had an attack that gave lasting immunity of high
degree, but maybe not enough to prevent or clear out a
suppressed infection he had carried for some years. Did his
immunity lay the foundation for the degenerative change in the
retina? If so does vaccination which, brings about comparable
condition not play a big role in the great dominance of
degenerative diseases of today? We say the virus or the germ
toxin must integrate with the cell’s functional structure to
bring about the loss of function and its sequel of tissue
degeneration and visa versa. Does not vaccination do the same?
If vaccination altered the steric set-up of the cell so fresh
entrance of the pathogen could not take hold, then the vaccine
must persist and do its damage to function. This would lead to
degenerative changes with gene effects.
Let us compare this type of immunity with the oxidative
protection used in Dr. Treiger’s Case 6A and in the
others. The scars did not form or they went away, as they were
replaced by functional parenchyma and the distant site of the
infection that brewed the toxin, be it scarlet fever, ordinary
pus infection, or some other factor that enters the etiology of
cancer, was also wiped out.
The cow experiments, a few of which are reported here, show
that the most vicious Staphylococci, etc., lose their
pathogenicity and maybe become faithful members of the great
Biological Economy after they are “cured” by oxidation
restoration facilities. Inferior oxidations, it will
appear as we go along, are the basis of disease.
RETINOBLASTOMA OF BOTH EYES (GLIOMA)
CASE No. 7
Rita Long was one year of age when the disease first made its
appearance. In less than a year the left eye was filled with a
tumor mass, irritated, swollen, and blind. The diagnosis was
made clinically by Dr. C., a noted Ophthalmologist, of Wichita,
Kansas, He removed the eye. The pathological report confirmed
his diagnosis of Retinoblastoma. The accompanying
document from the court records gives the gross and microscopic
pathology. This was in May 1934.
Within a year, the right eye showed the same changes. The same
surgeon observed the same disease here and so stated that it was
the same disease that had attacked the left eye. This is a well
known characteristic of this disease, Prof, Frohlich, of the
University of Michigan, testifying as an expert, stated that
there is nothing known to scientific medicine that can combat
this disease. Even when operated early it returns and kills. She
was taken to a renowned Ophthalmologist in Niles, Michigan, who
made the same diagnosis and referred her to us.
We examined her with Dr. H., a good specialist, and made the
same diagnosis. He found that one-third of the retina was
replaced by the neoplasm, including the optic disc. The eyeball
was bulging and distorted, the iris dilated and fixed. There was
no ability of the iris to move from its infiltrated attachments.
Behind the pupil was a yellowish, pinkish reflection to light,
showing that a tumor was present within. The area where the left
eye was removed was not healthy, but showed neoplastic
degeneration, though only in a minor degree. Both foci disturbed
her. At this time, November 25, 1935, she was over two years
old, and we gave her one cubic centimeter of the 10-(12)
solution of the SSR serially arranged Carbonyl groups with free
radical terminals by intramuscular injection. The irritation of
both foci soon left. Every third week, she exhibited a reaction
with general achiness, slight fever, and an aggravation of the
irritation of both foci. Each reaction lasted a few days and was
followed by improvement.
Examination on August 18, 1936, showed that all visible
pathology had been removed. Dr. H---- and Dr. W. made
Fundascopic examinations and found the retina normal, fully
restored. Since she lived so far away, we gave her another dose
for assurance, and no reaction thereto ever developed. She has
remained well to date with perfect vision in the right eye. She
went through her school courses and college at the head of her
classes, is now married and a healthy, happy woman. Reports made
in June, 1950, and June, 1953, and September, 1960 confirm her
full recovery.
This case along with the cases treated in Canada, all used the
same Treatment and are the only cases of Retinoblastoma ever
cured. And as Prof. Frohlich, of the University of Michigan
Department of Ophthalmology stated, “there is nothing known to
scientific medicine that can cope with this disease,” there is
no information available as to the changes in the retinoblastoma
cells during the recovery process. This is unfortunate for here
we have a tumor of a nerve tissue that is supposed not to be
able to reproduce in its normal state, even for a reconstructive
or compensatory purpose that does reproduce wildly with great
stubbornness, as a malignant tissue. In this case and the
others, the retina was restored with good vision performance. In
this case, the reconstruction was back to 20/20. The question
is, where did the new neurons come from, or were they some of
the tumor cells that were able to reconstruct functional
mechanisms, as soon as the FCG’s were liberated by the SSR? Thus
an undifferentiated tissue, probably because of FCG
inactivation, went malignant and then underwent normal
differentiation so as to be able to again function and take its
place in the organism’s economy, after its FCG’s was liberated.
This sequence of events calls for deep thought. It shows full
reversal of the malignant state.
EUNUCHOIDISM
CASE No. 8
J. S., at age 14, was subject to infections of the respiratory
tract and skin, presented marked obesity, female type shape, was
very dull mentally, was found to have an infantile penis and
undescended testicles. In fact, they had not even descended to
the canals. He had been under thyroid and pituitary hormone
therapy from the age of 10 in 1931, to age 14, in 1935, without
any improvement mentally, physically, or in his
resistance to infections. He was closely watched by Dr. S., a
relative, who reported that in this time the testicles had not
even entered the canals. Thus the disease was classified as the
type that is not helped by modern therapy and is permanent.
He received his dose of the SSR serial system of Carbonyl
groups in November 1935, and within three months, he lost
weight, his infections were gone, and the left testicle had
traversed the canal and was entering the scrotum. After the
sixth month, both testicles were in the scrotum, the penis was
developing, and the pubic hair distribution became masculine. He
grew taller, his hips reduced and his shoulders and jaw
developed to good proportions. He became very bright in his
schoolwork, soon making up for his backward position. He became
a good athlete. He entered the army and was soon promoted to a
corporal. He is married, and is raising a nice family of
children of his own. At the time this Testimony was given, he
was in the Army Law School where he was making splendid
progress. His boy and girl are physically normal and mentally
excellent. Thus his genes were re-established in normal line.
One may contrast 14 years of FCG insufficiency with a few months
of rescued FCG efficiency, placing him in the normal progression
of full health. *
* Photographs and a more complete case history of this patient
are included in The Birth of a Science.
CANCER OF THE TESTIS
CASE No. 9
Mr. T., age 38 in June 1925, when treated with SSR serial
system of Carbonyl groups with free radical terminals, his
testis had become malignant six months previous to its removal.
At this operation, no metastases were noted. The biopsy report
was “Medullary Carcinoma of the Testis.” Recurrence showed in
the groin and scrotum within six months, and another operation
revealed that the neoplasm had invaded the abdomen. Removal was
attempted and the microscopic report on the material removed
read again “Medullary Carcinoma of the Testis.” Recurrence was
not long in coming with more rapid spread of the disease. The
abdomen was again opened, but was found so inoperably involved
and a biopsy is all that was done. This biopsy was reported:
“Carcinoma, probably secondary to previous carcinoma of the
testis, as the cells are histologically similar.”
At this time he was rather emaciated, exhausted, and a general
cachexia called for an early termination, — a hopeless
prognosis. Through the intervention of Dr. Alpheus Hoyt, this
patient was given the SSR Reagent in June 1925. He regained his
health and all tumorous tissues disappeared. He remained well
thereafter and in 1946 he offered his report stating he was
perfectly well. This was 21 years after he received his
Treatment in the terminal phase of the disease. The preceding
case of anaplasia and this case of neoplasia were corrected by
the same Reagent, which restored good FCG function in both. Here
is food for thought.
MALIGNANT SYMPATHICOGONIOMA
CASE No. 10
Dr. Julian Baldor
John L., age 13 months, developed a tumor in the abdomen that
required an exploration on September 25th, 1951. A
retroperitoneal growth had infiltrated the region too thoroughly
to permit removal of any more than a biopsy. On October 6th,
examination revealed a visible bulging of the abdomen in the
umbilical region which we found on palpation to be the size of
an ordinary Florida grapefruit, about 10 to 15 cms. in its
diameters, firmly fixed to the surrounding structures. The
stools were bloody showing the intestine was invaded, and the
blood count agreed with his pallor, — 2,300,000 red cells with a
hemoglobin of 52%. The next day he received from Dr. Julian
Baldor an injection of the SSR Reagent, the serially arranged
system of Carbonyl groups with free radical ends, 2 cc’s 10-(12)
solution. Recovery began promptly and at the end of a year no
more tumor could be palpated on careful examination.
On May 5th, while in good health he was run over by an
automobile and sustained a broken leg and abdominal injuries.
While in the hospital for repairs, he was carefully examined by
the same surgeons who had removed the biopsy. They found him
free of any trace of palpable growth. He made a nice recovery
and is in excellent health still. His blood count on April 5,
1953, was 4,750,000 red cells, hemoglobin 87.5%. All the
documentation and facts of this case were put in the Florida
State Court Records by his surgeons in May 1953. His last report
was good health in 1958.
Discussion --- The case of Sympathicogonioma is of
interest not only because it is so very highly malignant, but
also that the mitotic mechanisms, which are now the malignant
cells, are separated from the nerve fibrilla, the functional
mechanism, as entities. The functional mechanism is entirely
shed or discarded and, of course, as an immature and unorganized
fibrillar syncitium. The malignant cells are practically only
made of nuclear material. It is unfortunate that periodic
sections could not have been taken of the tumor as it
normalized, to see if the neoplastic cells made any efforts to
form cytoplasm and fibrilla, as a recovery response. In the
Retinoblastoma case, the new neurons that restored the retina to
normal structure, probably had their origins in the neoplastic
cells, as new neurons are supposed never to be formed. In the
case of Optic atrophy, this assumption would also have to be
made with the conclusion that the new functional retinal neurons
were developed from remnants of atrophied cells that probably
were integrated with toxin as in symbiotic viral infections, and
the atrophy was never complete enough to prevent reconstruction
after the FCG was relieved of impediment.
That the FCG is the position that is stopped from functioning to
cause the pathology in each condition is quite evident.
Often a biopsy may reveal a tumor to be of grade one or two
malignancy, and after the tumor is removed the sections show it
is definitely a grade four, thus being changed by the
manipulations of the biopsy, the effect of the anesthetic, etc.
The case to be reviewed now is one that was exceedingly
anaplastic and a grade IV malignancy at biopsy, as the
examinations of the specimen by several leading pathologists and
many well-trained practitioners of medicine found. Some
microphotographs are submitted besides.
This case shows that the Survival chemistry, as established by
the high efficiency synthetic Carbonyl activity, may fade out in
ten years when the environmental conditions are adverse as they
were in this case, and the data should be compared with that of
the case that follows it, where a similar neoplasm of malignancy
was cured after the Synthetic Reagent was given, and still
remains cured 36 years afterward. Here the environmental
conditions were satisfactory, even though she was submitted to
the trauma of four natural perfect childbirths, and no return of
the neoplasm ever threatened. As in the neuroblastoma case just
reviewed, trauma alone is not sufficient to cause cancer or its
return. The anoxia may thus be established for a sufficient time
for the co-factor and the pathogen to make the integration.
However, with the improved Carbonyl function established by
oxidizing the pathogen away from the FCG system so that the FCG
is activated by a Carbonyl group, a sufficiently high O/R
potential is established to burn the pathogen, instead of
condensing with it. Still the Survival chemistry is a
comparative matter, as any other phase of life, and as this case
demonstrates.
It will be seen as we go along, as in the above case, that
anoxia alone is not sufficient to cause cancer, A CO-FACTOR,
be it a virus or another carcinogen, is required. Where
trauma is not concerned in causing the anoxia, toxic amines of
colonic origin may prepare the soil, and the response may not be
neoplastic, but a necrosis instead.
GRADE IV SQUAMOUS CELL CANCER OF THE CERVIX UTERI CASE
No. 11
Mrs. M. W. came under the care of Dr. L----, Dr. H----, and Dr.
D- -, of Medford, Oregon, in January, 1940. Dr. L---- examined
her and found that she had an enlarged and fixed uterus. She had
a cervix that protruded into the vagina and could be seen by a
vaginal examination with a speculum; a cervix which was
indurated and showed islands of apparent new growth. A diagnosis
of far advanced cancer of the uterus was made clinically. Dr.
L---- reported that her cancerous condition would probably, if
untreated, end her life within a year. That because of the
fixation of the uterus and the involvement of the adnexia, it
was his opinion, that it was not a surgical case, as surgery
would have had to be too extensive. It was too late for that
sort of thing. The case had already entered the cachexia stage
as Doctor L----’s Testimony reveals:
“She had lost 30 pounds in six weeks, was complaining of
general weakness and had a rather poor color at the time.” As
Ewing states: “Characteristic cachexia in uterine cancer
develops in the terminal stage of the generalized disease, but
when the lesion is localized in the pelvis, cachexia is
missing.”
Dr. L---- gave the SSR Treatment, 2 cc.’s of the 10-(12)
solution on March 20, 1940, December 30, 1940, and October,
1941. His examination made a year and a half later, in the
summer of 1942, found that: “She had gained weight, she had
gained color, and improved in general appearance. The mass in
the abdomen had subsided to the extent that he could no longer
palpate it. The appearance of the cervix by examination with the
speculum appeared normal.”
Before giving the SSR injection she was placed in the Medford
Hospital, and there Dr. L---- removed a specimen from the cervix
for microscopic study. The slide was very carefully prepared by
Dr. Green, the hospital’s Pathologist, and diagnosed by him as
squamous cell cancer of the cervix uteri. But the case was so
interesting that he sent the slide to Professor Hunter, the
Pathologist at the University, who established the grade of
malignancy. Dr. Hunter, in his letter to Dr. Green on January
27, 1940, wrote: “I see a decidedly invasive and anaplastic
carcinoma which occupied well over half of the tissue . . . I
would if asked to do so, grade this one as a IV.”
